Hungarian 
The incidence of neurodegenerative diseases and cyanobacterial blooms is concomitantly increasing worldwide.
This new review evaluates different neuropathological mechanisms of BMAA at molecular and cellular levels and compares the related studies to provide some useful recommendations. Additionally, the structure and properties of BMAA as well as its microbial origin, especially by gut bacteria, are also briefly covered. Unlike previous reviews, the researchers hypothesize the possible neurotoxic mechanism of BMAA through iron overload. The BMAA may mediate in ferroptosis which results in the death of neuronal cells. BMAA-induced Fe overload can also cause cell damage in alternative ways (lipids, proteins, DNA and carbohydrates). Iron overload may also cause expression and aggregation of α-Syn (PD hallmark), and increase Aβ density and senile plaques formation (AD hallmark).
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